Neurorescue Effects of Frondoside A and Ginsenoside Rg3 in C. elegans Model of Parkinson’s Disease

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dc.contributor.authorPawanrat Chalorak
dc.contributor.authorTanatcha Sanguanphun
dc.contributor.authorTanapol Limboonreung
dc.contributor.authorKrai Meemon
dc.date.accessioned2026-05-08T19:15:14Z
dc.date.issued2021-8-10
dc.description.abstractmodel through suppressing apoptosis mediators and stimulating antioxidant enzymes. In addition, FA could attenuate α-synuclein aggregation through the protein degradation process.
dc.identifier.doi10.3390/molecules26164843
dc.identifier.urihttps://dspace.kmitl.ac.th/handle/123456789/14919
dc.publisherMolecules
dc.subjectSeaweed-derived Bioactive Compounds
dc.subjectAldose Reductase and Taurine
dc.subjectGenomics, phytochemicals, and oxidative stress
dc.titleNeurorescue Effects of Frondoside A and Ginsenoside Rg3 in C. elegans Model of Parkinson’s Disease
dc.typeArticle

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