Limboonreung, Tanapol

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Limboonreung, Tanapol
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tanapol.li@kmitl.ac.th

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    Item type:Publication,
    Corrigendum to “Diterpene glycosides from Holothuria scabra exert the α-synuclein degradation and neuroprotection from α-synuclein-mediated neurodegeneration in C. elegans model” [J. Ethnopharmacol. 279 (2021) 114347](S0378874121005766)(10.1016/j.jep.2021.114347)
    (2023-01-30)
    Chalorak, Pawanrat
    ;
    Sornkaew, Nilubon
    ;
    Manohong, Preeyanuch
    ;
    Niamnont, Nakorn
    ;
    Malaiwong, Nawapaht
    The authors regret that two images on Figs. 6A and 7A were inadvertently duplicated during the assembly of the figures. The authors reviewed the original data and reorganized the correct representative images. The correct Figs. 6A and 7A are shown below. As described in the article, the images displayed in the figures were representative from each experimental group which were taken from the confocal microscopy, while the data represented in the graph were analysed from many images collected by using fluorescence microscopy. Therefore, the authors confirmed that the corrections of the images made in this erratum do not affect the figure legends, results, and conclusion of the study.[Formula presented][Formula presented] The authors would like to apologise for any inconvenience caused.
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    Diterpene glycosides from Holothuria scabra exert the α-synuclein degradation and neuroprotection against α-synuclein-Mediated neurodegeneration in C. elegans model
    (2021-10-28)
    Chalorak, Pawanrat
    ;
    Sornkaew, Nilubon
    ;
    Manohong, Preeyanuch
    ;
    Niamnont, Nakorn
    ;
    Malaiwong, Nawaphat
    Ethnopharmacological relevance: Holothuria (Metriatyla) scabra Jaeger (H. scabra), sea cucumber, is the marine organism that has been used as traditional food and medicine to gain the health benefits since ancient time. Although our recent studies have shown that crude extracts from H. scabra exhibited neuroprotective effects against Parkinson's disease (PD), the underlying mechanisms and bioactive compounds are still unknown. Aim of the study: In the present study, we examined the efficacy of purified compounds from H. scabra and their underlying mechanism on α-synuclein degradation and neuroprotection against α-synuclein-mediated neurodegeneration in a transgenic Caenorhabditis elegans PD model. Material and methods: The H. scabra compounds (HSEA-P1 and P2) were purified and examined for their toxicity and optimal dose-range by food-clearance and lifespan assays. The α-synuclein degradation and neuroprotection against α-synuclein-mediated neurodegeneration were determined using transgenic C. elegans model, Punc-54